Equine protozoal myeloencephalitis epm essay
If the animal improves, then it is concluded that the horse did have EPM. All commonly used tests are based on detection of antiprotozoal antibodies in serum, CSF, or both. Any cranial nerve nucleus may be involved, and there may be seizures, visual deficits including abnormal menace responses, or behavioral abnormalities. The Cornell Veterinarian. Horses on farms where previous cases of EPM have been diagnosed are at an increased risk. Focal muscle atrophy is not common. This disease can be difficult for owners and veterinarians to diagnose and treat. Quarter Horses, other breeds, ponies, and drafts had a lower incidence rate. With all types of life-threatening disease, it is important that you recognize, diagnose and treat early for the best possible outcome. Horses contract EPM from contaminated feed or water, so killing other wildlife is counterproductive; keeping a clean barn and covering feed is more effective in preventing the disease. Another important issue to keep in mind is that some horses that have responded favorably to treatment may be affected again, often with similar signs, at a later time.
Keep your feed in sealed or closed containers. Untidy areas with spilled feed invites unwanted guests such as opossums.
Causes[ edit ] Life cycle of S. The natural host s of this organism have not yet been identified. A second possibility is that some other factor or agent predisposes horses to develop disease or causes an existing infection with Sarcocystis neurona to change from a latent to a lethal condition.
Early signs of EPM such as stumbling and frequent interference between limbs can be confused with lameness. There are no false positives with this test. The disease usually begins insidiously but may present acutely and be severe at onset.
If separate sources of water are available for horses and wildlife so horses drink separately from opossums, there is lessened chance of infection.
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